Antianxiety Medications etiketine sahip kayıtlar gösteriliyor. Tüm kayıtları göster
Antianxiety Medications etiketine sahip kayıtlar gösteriliyor. Tüm kayıtları göster

GETTING THE ADDICT TO AGREE TO INVOLVE HIS FAMILY OF ORIGIN

16 Mayıs
IN CONTRAST to the analysis presented thus far concerning the functional value of addiction in families,6T.'"• [".15"' 1" 54' v" a number of investigators have given evidence that the family can also be important in the rehabilitation of the addict. When family members are involved in the treatment process, the system can be changed toward helping the abusing member overcome his addiction rather than serving as a force that maintains it. To this point, Eldred and Washington42 found in interviews with 158 heroin addicts that the people who the patients thought would be most helpful to them in their attempts to give up drugs were the members of their families of origin or their in-laws; second and third choices were an opposite-sex partner and the patient himself. A group of 462 heroin addicts interviewed by MACRO Systems researchers91 reported that the family was second only to treatment (70.9% vs. 79.6%) as the influence they perceived as most important in changing their lives. Finally, a 5-year follow-up study of narcotics addicts by Levy92 found that patients who successfully over-came their drug habits most often had family support.
Studies of the effectiveness of family treatment have shown this to be a promising approach with alcoholism and many other symp-toms, and family therapies for drug problems have been gaining


This chapter is an expansion and revision of a paper by the first four authors entitled "Engaging 'Resistant' Families in Treatment: I. Getting the Drug Addict to Recruit His Family Members'. and is reprinted with permission from the International Journal of the Addictions, 1980, 15, 1069-1089. (1) Marcel Dekker, Inc. momentum in recent years. t" In fact, a 1976 national survey of 2012 drug programs by Coleman and Davis3" indicated that 93% were providing some kind of family services for at least a portion of their clients—in many cases, family therapy. Consequently, if there is a validity to such efforts, it is important to be able to induct family members into the treatment program. This chapter presents tech-niques pertaining to the initial facet of this process, that is, dealing with the addict in recruiting his family.*
As noted in the literature review in Chapter 5, authors who have dealt with this matter have noted how difficult it can be to bring family members of compulsive drug abusers into treatment. Most family members, especially parents, generally refuse to become involved. This is particularly interesting in view of the aforementioned evidence that the majority of drug addicts—especially those who use opiates—maintain close ties to their families of origin. If they do not live with one or both parents they may reside nearby and be in frequent contact (see Appendix A for a review of these studies). Thus it becomes all the more important that methods for effectively involving members of addicts' families in the treatment process be developed.
The AFP research design called for a Family Evaluation Session (see Appendix C), which included at least the addict, both his parents or parent surrogates (e.g., stepmother, mother's boyfriend), and any siblings living nearby. This session was required before treatment could proceed. (Although not included in the Family Evaluation Session, spouses of married addicts were usually involved in the therapy that followed it.) Obviously, this put considerable pressure on us to succeed in our recruiting efforts. The general procedure was for the person responsible for inducting the family to function as both drug counselor and family therapist. In approximately 80% of the cases we were able to obtain cooperation from the addict toward including his family, and in 88% of these we were able to get the family—including both parents or parent surrogates—to physically appear at the treatment site together. In other words, two-thirds of the subset of families we were unable to recruit occurred because the addict
*it should be noted that the primary emphasis of this chapter is upon young adult addicts. Adolescents may also manifest resistance to family involvement, However, the way the therapist handles the process may differ when encountering an adolescent versus a young adult, Some pointers and strategies for getting adoles‑
cents to agree to having their families become involved in therapy are presented in Chapter 13. would not allow us to contact his family, which underscores the emphasis given to the initial patient interview in this chapter.
JOHN M. VAN DEUSEN/M. DUNCAN STANTON/
SAMUEI. M. SCUTT/THOMAS C. TODD/DAVID T. MOWATT

DANGEROUS INTERACTIONS WITH OTHER DRUGS

10 Mayıs

DANGEROUS INTERACTIONS WITH OTHER DRUGS

OTHER DRUGS

               Sedatives

Clearly the most dangerous drugs to mix with alcohol are other sedatives, or "downers," such as phenobarbital and pentobarbital. The depressing  effects of alcohol on brain function combined with the effects of the bar­biturates can cause extreme impairment, unconsciousness, or even death. One of the most famous cases in medical ethics was that of a young woman, Karen Ann Quinlan, who drank alcohol in combination with Quaaludes (methaqualone—a powerful sedative drug) and went into a coma from which she never recovered. This tragic case gained national attention because it raised the issue of whether a person should be removed from life-support machines after it becomes clear that he or she will never recover from a vegetative state.




Although few people take alcohol-sedative combinations severe enough to cause coma or death, the combination of even relatively low doses of alcohol and sedatives can be dangerous, powerfully impairing the ability to think clearly, make good decisions, or drive a car. A person who is nor­mally able to perform these tasks perfectly well at the end of an evening after having had three or four beers over the course of several hours might find that he is totally unable to perform them if even a small dose of seda­tives is added to the mix. The effects of the alcohol may be totally unex­pected in the presence of the other sedative drug.

Antianxiety Medications
OTHER DRUGS

Antianxiety medications, such as Valium, Librium, and so forth, fall into the general category called benzodiazepines and are used to treat anxiety, sleep disturbances, and seizures. They are also used to treat alcohol-with­drawal symptoms in detoxification clinics, These drugs are sedating and may cause severe drowsiness in the presence of alcohol, increasing the risk of household and automobile accidents.
Antibiotics
In combination with acute doses of alcohol, some antibiotics can cause nausea, vomiting, headache, or even convulsions (seizures). Among the potentially dangerous ones are Furoxone (furazolidone), Grisactin (gris­eofulvin), Flagyl (metronidazole), and Atabrine (quinacrine).
Anticoagulants (Blood Thinners)
Warfarin (Coumadin) is prescribed to decrease the blood's ability to clot.
Alcohol increases the availability of warfarin in the body and increases
the risk of dangerous bleeding. But in chronic drinkers, warfarin's action
is decreased, lessening these patients' protection from the consequences of blood-clotting disorders.
Antidepressants
Many people who are depressed use alcohol, and many alcoholics are also depressed. So, it is quite common for people to use alcohol with antide­pressant drugs. Alcohol increases the sedative effects of the tricyclic anti­depressants such as Elavil (amitriptyline). This impairs both mental and physical skills such as those necessary for driving. Chronic drinking appears to increase the action of some tricyclic antidepressants and decrease the action of others. Anyone who is on antidepressants should
consult closely with her doctor about how her medication reacts with alcohol.
Ant/diabetic Medications
Orinase (tolbutamide) is given orally to help lower blood sugar in diabetic patients. Acute alcohol drinking prolongs the action of this drug, and chronic drinking decreases its availability in the body. Alcohol can also cause nausea and headache when taken with some drugs of this class.
Antihistamines
Antihistamines such as Benadryl (diphenhydramine) are available with­out a prescription and are used to treat allergic symptoms and sometimes insomnia. They have sedative effects that may be intensified by alcohol, increasing the probability of accidents. In older persons these drugs can
cause excessive dizziness and sedation, and their combination with alco­hol may be particularly dangerous.
Ant/psychotic Medications
Drugs such as Thorazine (chlorpromazine) are used to treat psychotic symptoms such as delusions and hallucinations. Acute alcohol drinking can increase the sedative effects of these drugs, resulting in impaired coordination and potentially fatal suppression of breathing.
Antiseizure Medications
OTHER DRUGS

One of the most widely used drugs prescribed to treat epilepsy (seizures) is Dilantin (phenytoin). Acute alcohol drinking increases the availability of Dilantin in the body and increases the probability of side effects. Chronic drinking may decrease the availability of Dilantin, dangerously hampering its effectiveness and increasing the patient's risk of seizures.
Heart Medications
There are many medications used to treat disease of the heart or circula­tory system. Acute alcohol drinking can interact with some of these to cause dizziness or fainting upon standing up. These drugs include the angina medicine nitroglycerin and the blood pressure medication Apre­soline. In addition, chronic alcohol drinking reduces the effectiveness of the blood pressure medication Inderal (propranoloh.
Narcotic Pain Relievers
These drugs (e.g., morphine, Darvon, codeine, Demerol) are prescribed for moderate to severe pain, such as after surgery or dental work. The combi­nation of any of these drugs with alcohol magnifies the sedative effects of both, increasing the risk of death from overdose. This is one of the most common drug combinations to cause accidental overdose deaths.
Nonnarcotic Pain Relievers
Some nonprescription pain relievers such as aspirin, Advil, and Aleve can cause stomach bleeding and prevent the blood from clotting normally Alcohol can worsen these side effects. In addition, aspirin may increase the availability of alcohol within the body, thereby increasing the intoxi­cating effect of a given drink. As we stated before, the combination of Tylenol (acetaminophen) and alcohol can result in the formation of chem­icals that can cause liver damage. This can occur even when the pain reliever is used in recommended doses and even if it is taken after drinking as a treatment for hangover.

CYNTHIA KUHN